Deoxycorticosterone upregulates PDS (Slc26a4) in mouse kidney: Role of pendrin in mineralocorticoid-induced hypertensionVERLANDER, Jill W; HASSELL, Kathryn A; ROYAUX, Ines E et al.Hypertension (Dallas, Tex. 1979). 2003, Vol 42, Num 3, pp 356-362, issn 0194-911X, 7 p.Article

Expression of PDS/Pds, the Pendred syndrome gene, in endometriumSUZUKI, Koichi; ROYAUX, Ines E; KOHN, Leonard D et al.The Journal of clinical endocrinology and metabolism. 2002, Vol 87, Num 2, pp 938-941, issn 0021-972XArticle

Reduced ENaC protein abundance contributes to the lower blood pressure observed in pendrin-null miceYOUNG HEE KIM; PECH, Vladimir; SPENCER, Kathryn B et al.American journal of physiology. Renal physiology. 2007, Vol 62, Num 4, issn 1931-857X, F1314-F1324Article

NaCl restriction upregulates renal Slc26a4 through subcellular redistribution: Role in Cl^- conservationWALL, Susan M; YOUNG HEE KIM; STANLEY, Lorraine et al.Hypertension (Dallas, Tex. 1979). 2004, Vol 44, Num 6, pp 982-987, issn 0194-911X, 6 p.Article

Targeted disruption of mouse Pds provides insight about the inner-ear defects encountered in Pendred syndromeEVERETT, Lorraine A; BELYANTSEVA, Inna A; NOBEN-TRAUTH, Konrad et al.Human molecular genetics (Print). 2001, Vol 10, Num 2, pp 153-161, issn 0964-6906Article